KLOW enables researchers to study the intersection of inflammation control and tissue repair. KPV suppresses excessive inflammatory signaling (NF-κB inhibition) while BPC-157, TB500, and GHK-Cu promote three different tissue repair mechanisms. The hypothesis under investigation is whether controlling inflammation simultaneously with promoting repair produces better outcomes than either approach alone.1
IMMUNE
KLOW
$129.99
KLOW is a composite research peptide blend comprising BPC-157, thymosin beta-4, GHK-Cu and KPV. Supplied as a high-purity lyophilized powder, it supports in vitro exploration of angiogenesis, extracellular matrix turnover, cytoskeletal organization, and inflammatory signaling using complementary pathways derived from the component molecules. For laboratory research only, and controlled assays.
QuantityStandard research vial
PuritySee COA
Free shipping on orders over $200
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Product Overview
KLOW is an OSYRIS Health immune/recovery stack combining four compounds: KPV (anti-inflammatory tripeptide), BPC-157 (growth factor modulator), TB500 (actin-mediated cell migration), and GHK-Cu (copper peptide, gene expression modulator). This is the most comprehensive multi-compound formulation in the OSYRIS catalog, targeting both inflammatory signaling and tissue repair through four distinct mechanisms.
The stack is designed for research protocols investigating the interplay between immune regulation and tissue repair — an intersection that is increasingly recognized as critical to understanding healing processes, as inflammation both initiates and can impair repair depending on its intensity and duration.
Research Applications
Mechanism and Experimental Context
KPV provides direct NF-κB inhibition, while BPC-157 has independent anti-inflammatory properties through NO system modulation. The combination allows researchers to study whether targeting inflammation through multiple pathways produces more comprehensive immune modulation than single-pathway intervention.
The three repair components (BPC-157 growth factors, TB500 cell migration, GHK-Cu gene expression + collagen synthesis) represent the most complete set of tissue repair mechanisms available in a single formulation, enabling multi-variable tissue biology research protocols.
Explore Related Compounds
Keep the Research Trail Moving
Referenced Studies
Source Literature
[1]
Dalmasso G, et al. "KPV attenuates inflammatory colitis." PLoS ONE, 2008. PubMed: PMID 18941502
[2]
Sikiric P, et al. "BPC 157: novel therapy in gastrointestinal tract." Current Pharmaceutical Design, 2011. PubMed: PMID 21861804
Certificate of Analysis
Batch Documentation
Current published batch documentation is surfaced on-page whenever the provider exposes a public COA asset.
Frequently Asked Questions
Questions About KLOW
KLOW combines KPV + BPC-157 + TB500 + GHK-Cu in a single vial. It targets both immune regulation (KPV) and tissue repair (BPC-157, TB500, GHK-Cu) for integrated research protocols.
KPV (the lead ingredient) is an anti-inflammatory peptide that inhibits NF-κB. The stack's primary research application is the intersection of immune regulation and tissue repair — making Immune & Host Defense the correct category.
KLOW focuses on immune + recovery (KPV + BPC-157 + TB500 + GHK-Cu). GLOW focuses on aesthetics (BPC-157 + GHK-Cu + TB500). KLOW adds KPV for anti-inflammatory research. GLOW omits KPV and is categorized under Aesthetics.
Yes. KPV, BPC-157, TB500, and GHK-Cu are all available as individual products.
Lyophilized at 2-8°C or -20°C. Protect from light.
Each component is independently tested via HPLC and LC-MS. COA downloadable on this page.
No. Research compound sold exclusively for laboratory research.
It targets four distinct mechanisms: NF-κB inhibition (KPV), growth factor modulation (BPC-157), actin-mediated cell migration (TB500), and gene expression modulation with copper delivery (GHK-Cu). No other OSYRIS product covers this many pathways.
Every Batch Tested by an Independent Lab
We publish the Certificate of Analysis for every product. See our full testing process.